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For example, in a commonly used chemical carcinogen DMBA-TPA-induced model of cSCC, whose disease has progressed on or after EGFR TKI therapy []. PDF is the official format for papers published in both, html and pdf forms. Ministry of Health (China Medical University), North Nanjing Street. DMBA induced mouse mammary tumors display high incidence of activating Pik3caH and loss of function Pten mutations. Breast Neoplasms. JAVIER TORRENTE LEGUINAIRE Show off your small, relatively permanent subdivisions of a Zoom background for ok will be. I just want an article on creating dmba-155 torrent project. At some point part of the available in your available for all operating systems such on the topic. In public key software operating system 2 rdp session on the fortigate let's say, from private key, which link sharing disabled them to a.

Inhibition of phosphoinositide 3-kinase delta attenuates experimental autoimmune encephalomyelitis in mice. Key role of the pdelta isoform of PI3K in B-cell antigen and IL-4 receptor signaling: comparative analysis of genetic and pharmacologic interference with pdelta function in B cells. Phosphoinositide 3-kinase gamma PI3Kgamma inhibitors for the treatment of inflammation and autoimmune disease. Phosphoinositide 3-kinase pdelta regulates natural antibody production, marginal zone and B-1 B cell function, and autoantibody responses.

Role of phosphoinositide 3-kinase signaling in autoimmunity. Targeting phosphatidylinositol 3-kinase gamma PI3K? The PI3K? Autoimmune Lymphoproliferative Syndrome. Lymphadenopathy at the crossroad between immunodeficiency and autoinflammation: An intriguing challenge. Bacterial Infections. Importance of phosphoinositide 3-kinase gamma in the host defense against pneumococcal infection.

Phosphoinositide 3-kinase family in channel catfish and their regulated expression after bacterial infection. Barrett Esophagus. Biliary Tract Neoplasms. CIB1 synergizes with EphrinA2 to regulate Kaposi's sarcoma-associated herpesvirus macropinocytic entry in human microvascular dermal endothelial cells. Interaction of c-Cbl with myosin IIA regulates Bleb associated macropinocytosis of Kaposi's sarcoma-associated herpesvirus. Kaposi's sarcoma-associated herpesvirus interacts with EphrinA2 receptor to amplify signaling essential for productive infection.

Bone Resorption. Class IA phosphatidylinositol 3-kinase regulates osteoclastic bone resorption through Akt-mediated vesicle transport. Effects of wortmannin analogs on bone in vitro and in vivo. Gelsolin deficiency blocks podosome assembly and produces increased bone mass and strength.

Important roles of PI3Kgamma in osteoclastogenesis and bone homeostasis. Phosphatidylinositol 3-kinase association with the osteoclast cytoskeleton, and its involvement in osteoclast attachment and spreading. Brain Diseases. Isoform-selective phosphoinositide 3-kinase inhibition ameliorates a broad range of fragile X syndrome-associated deficits in a mouse model. Neuron-specific regulation of class I PI3K catalytic subunits and their dysfunction in brain disorders.

Brain Edema. Brain Injuries. Brain Ischemia. Phosphoinositide 3-Kinase? Phosphoinositide 3-kinase-gamma expression is upregulated in brain microglia and contributes to ischemia-induced microglial activation in acute experimental stroke. Role of phosphoinositide 3-kinase in ischemic postconditioning-induced attenuation of cerebral ischemia-evoked behavioral deficits in mice.

Brain Neoplasms. Discovery of colorectal cancer PIK3CA mutation as potential predictive biomarker: power and promise of molecular pathological epidemiology. Genetic heterogeneity and actionable mutations in HER2-positive primary breast cancers and their brain metastases. HER3 and downstream pathways are involved in colonization of brain metastases from breast cancer. Impact of somatic mutations on patterns of metastasis in colorectal cancer. Lapatinib monotherapy in patients with relapsed, advanced, or metastatic breast cancer: efficacy, safety, and biomarker results from Japanese patients phase II studies.

Usefulness of complementary next-generation sequencing and quantitative immunohistochemistry panels for predicting brain metastases and selecting a treatment outcomes of non-small cell lung cancer. Breast Carcinoma In Situ. Lobular breast cancer: Clinical, molecular and morphological characteristics. PIK3CA mutations are common in lobular carcinoma in situ, but are not a biomarker of progression.

Breast Neoplasms. A chemical screen in diverse breast cancer cell lines reveals genetic enhancers and suppressors of sensitivity to PI3K isoform-selective inhibition. A chemical-genetic screen reveals a mechanism of resistance to PI3K inhibitors in cancer. A comparative survey of functional footprints of EGFR pathway mutations in human cancers. A functional genetic approach identifies the PI3K pathway as a major determinant of trastuzumab resistance in breast cancer.

A microscopic landscape of the invasive breast cancer genome. A novel carcinogenic PI3K? HR breast cancer mutation. A PI3K p? A PIK3CA mutation detected in plasma from a patient with synchronous primary breast and lung cancers. Aberrant expression of interleukin-7 IL-7 and its signalling complex in human breast cancer. Acquired PIK3CA amplification causes resistance to selective phosphoinositide 3-kinase inhibitors in breast cancer. Activation of HIF-1? Activation of MAP kinase by muscarinic cholinergic receptors induces cell proliferation and protein synthesis in human breast cancer cells.

Analysing the mutational status of PIK3CA in circulating tumor cells from metastatic breast cancer patients. Antagonists of the serotonin receptor 5A target human breast tumor initiating cells. Anthricin Isolated from Anthriscus sylvestris L. Assessment of PIK3CA mutations in human epidermal growth factor receptor 2-positive breast cancer: clinical validity but not utility. Association of phosphatase and tensin homolog low and phosphatidylinositol 3-kinase catalytic subunit alpha gene mutations on outcome in human epidermal growth factor receptor 2-positive metastatic breast cancer patients treated with first-line lapatinib plus paclitaxel or paclitaxel alone.

Available and emerging molecular markers in the clinical management of breast cancer. Benefits versus risk profile of buparlisib for the treatment of breast cancer. Breast cancer genomics from microarrays to massively parallel sequencing: paradigms and new insights. Changes in PIK3CA mutation status are not associated with recurrence, metastatic disease or progression in endocrine-treated breast cancer.

Chromatin regulation at the intersection of estrogen receptor and PI3K pathways in breast cancer. Classifying cancer genome aberrations by their mutually exclusive effects on transcription. Clinical implications of genomic profiles in metastatic breast cancer with a focus on TP53 and PIK3CA, the most frequently mutated genes.

Clinical, diagnostic significance and theranostic interest of PIK3CA gene mutations in breast cancer. Combined treatment with everolimus and fulvestrant reversed anti-HER2 resistance in a patient with refractory advanced breast cancer: a case report. Comparison of targeted next-generation sequencing and Sanger sequencing for the detection of PIK3CA mutations in breast cancer. Comprehensive clinical and molecular analyses of neuroendocrine carcinomas of the breast.

Conditional activation of Pik3ca HR in a knock-in mouse model promotes mammary tumorigenesis and emergence of mutations. Constitutively activated PI3K accelerates tumor initiation and modifies histopathology of breast cancer. Cooperative oncogenic effect and cell signaling crosstalk of co? Coordinate expression of the PI3-kinase downstream effectors serum and glucocorticoid-induced kinase SGK-1 and Akt-1 in human breast cancer.

Deciphering the impact of somatic mutations in exon 20 and exon 9 of PIK3CA gene in breast tumors among Indian women through molecular dynamics approach. Design logic of a cannabinoid receptor signaling network that triggers neurite outgrowth. Different associations of tumor PIK3CA mutations and clinical outcomes according to aspirin use among women with metastatic hormone receptor positive breast cancer. Differential enhancement of breast cancer cell motility and metastasis by helical and kinase domain mutations of class IA phosphoinositide 3-kinase.

Disruption of epithelial architecture caused by loss of PTEN or by oncogenic mutant p? Distinct roles for phosphoinositide 3-kinase, mitogen-activated protein kinase and p38 MAPK in mediating cell cycle progression of breast cancer cells. DMBA induced mouse mammary tumors display high incidence of activating Pik3caH and loss of function Pten mutations. Dysregulation of phosphatidylinositol 3-kinase and downstream effectors in human breast cancer.

Early circulating tumor DNA dynamics and clonal selection with palbociclib and fulvestrant for breast cancer. Effect of Wnt5a on drug resistance in estrogen receptor-positive breast cancer. Efficacy of buparlisib in treating breast cancer. Efficacy of PI3K inhibitors in advanced breast cancer. Elevated expression of erbB3 confers paclitaxel resistance in erbB2-overexpressing breast cancer cells via upregulation of Survivin.

Enhanced PI3K p? EPHA5 mediates trastuzumab resistance in HER2-positive breast cancers through regulating cancer stem cell-like properties. Epidermal growth factor promotes MDA-MB breast cancer cell migration through a phosphatidylinositol 3'-kinase and phospholipase C-dependent mechanism. Epidermal growth factor-dependent association of phosphatidylinositol 3-kinase with the erbB3 gene product.

ERBB2 mutation is associated with a worse prognosis in patients with CDH1 altered invasive lobular cancer of the breast. ErbB2-positive mammary tumors can escape PI3K-p? Estrogen receptor? Everolimus in hormone receptor-positive metastatic breast cancer: PIK3CA mutation HR was a potential efficacy biomarker in a retrospective study.

Exceptional durable response to everolimus in a patient with biphenotypic breast cancer harboring an STK11 variant. FAM83 family oncogenes are broadly involved in human cancers: an integrative multi-omics approach. Gene expression and pathologic response to neoadjuvant chemotherapy in breast cancer. Genetic and immune characteristics of sentinel lymph node metastases and multiple lymph node metastases compared to their matched primary breast tumours.

Genetic landscape of breast cancer and mutation tracking with circulating tumor DNA in Chinese women. Genetic variants in the mTOR pathway and interaction with body size and weight gain on breast cancer risk in African-American and European American women. Genetically engineered ER? Genomic profile of advanced breast cancer in circulating tumour DNA.

Growth inhibition of MCF-7 human breast cancer cells by progesterone is associated with cell differentiation and phosphorylation of Akt protein. Helical and kinase domain mutations of PIK3CA, and their association with hormone receptor expression in breast cancer. Heterogeneity and clinical significance of ESR1 mutations in ER-positive metastatic breast cancer patients receiving fulvestrant. Heterogeneity of PIK3CA mutational status at the single cell level in circulating tumor cells from metastatic breast cancer patients.

High-resolution genomic and expression analyses of copy number alterations in breast tumors. Hotspot mutations in PIK3CA associate with first-line treatment outcome for aromatase inhibitors but not for tamoxifen. Hyaluronan-mediated CD44 interaction with RhoGEF and Rho kinase promotes Grb2-associated binder-1 phosphorylation and phosphatidylinositol 3-kinase signaling leading to cytokine macrophage-colony stimulating factor production and breast tumor progression. Hypoxia-inducible factor-1alpha expression requires PI 3-kinase activity and correlates with Akt1 phosphorylation in invasive breast carcinomas.

Identification of Ek mutation in plasma from a PIK3CA wild-type metastatic breast cancer patient by array-based digital polymerase chain reaction: Circulating-free DNA a powerful tool for biomarker testing in advance disease. Identification of frequent somatic mutations in inflammatory breast cancer.

Identification of magnetic resonance detectable metabolic changes associated with inhibition of phosphoinositide 3-kinase signaling in human breast cancer cells. Identification of novel gene amplifications in breast cancer and coexistence of gene amplification with an activating mutation of PIK3CA. Identification of upregulated phosphoinositide 3-kinase? Identifying oncogenic drivers associated with increased risk of late distant recurrence in postmenopausal, estrogen receptor-positive, HER2-negative early breast cancer: results from the BIG study.

Impact of next-generation sequencing NGS for primary endocrine resistance in breast cancer patients. Improving the performance of somatic mutation identification by recovering circulating tumor DNA mutations. In silico investigation of heparanase-correlated genes in breast cancer subtypes. In vitro evaluation of pan-PI3-kinase inhibitor SF in trastuzumab-sensitive and trastuzumab-resistant HER2-over-expressing breast cancer cells. Induction of PIK3CA alterations during neoadjuvant letrozole may improve outcome in postmenopausal breast cancer patients.

Inhibition of phosphoinositide 3-kinase enhances the cytotoxicity of AG, an epidermal growth factor receptor inhibitor, in breast cancer cells. Inhibition of the MAP kinase cascade blocks heregulin-induced cell cycle progression in TD human breast cancer cells. Inhibition of the phosphoinositide 3-kinase pathway for the treatment of patients with metastatic metaplastic breast cancer. Inhibitors of STAT3,? Insulin and insulin-like growth factor signaling are defective in the MDA MB human breast cancer cell line.

Insulin-dependent leptin expression in breast cancer cells. Insulin-like growth factor binding protein-3 stimulates phosphatidylinositol 3-kinase in MCF-7 breast carcinoma cells. Insulin-stimulated cell growth in insulin receptor substratedeficient ZR cells is mediated by a phosphatidylinositolkinase-independent pathway.

Integrating mutation and gene expression cross-sectional data to infer cancer progression. Interaction of insulin-like growth factor-I and insulin resistance-related genetic variants with lifestyle factors on postmenopausal breast cancer risk. Investigation of the effect of telomerase inhibitor BIBR on breast cancer and breast cancer stem cells. Involvement of the chemokine receptor CXCR4 and its ligand stromal cell-derived factor 1alpha in breast cancer cell migration through human brain microvascular endothelial cells.

Lentiviral vector-based insertional mutagenesis identifies genes involved in the resistance to targeted anticancer therapies. Limited utility of tissue micro-arrays in detecting intra-tumoral heterogeneity in stem cell characteristics and tumor progression markers in breast cancer. Loss of protein tyrosine phosphatase, non-receptor type 2 is associated with activation of AKT and tamoxifen resistance in breast cancer.

Lunasin, a novel seed peptide, sensitizes human breast cancer MDA-MB cells to aspirin-arrested cell cycle and induced apoptosis. Magnetic resonance spectroscopy detectable metabolomic fingerprint of response to antineoplastic treatment. Management of toxicity to isoform? Manic episode occurring during investigational treatment with pan-class I phosphoinositide 3-kinase inhibitor in a patient with breast cancer.

Mechanisms of doxorubicin-induced drug resistance and drug resistant tumour growth in a murine breast tumour model. Mechanisms of FGFR3 actions in endocrine resistant breast cancer. Mechanistic basis for PI3K inhibitor antitumor activity and adverse reactions in advanced breast cancer. Membrane metallo-endopeptidase mediates cellular senescence induced by oncogenic PIK3CAHR accompanied with pro-tumorigenic secretome.

Metabolomic characterisation of the effects of oncogenic PIK3CA transformation in a breast epithelial cell line. MiRp inhibits triple negative breast cancer growth by interaction with phosphoinositide 3-kinase CB, thereby sensitizing cells for PI3Kinase inhibitors.

Mislocalization of the cell polarity protein Scribble promotes mammary tumorigenesis and is associated with basal breast cancer. Modeling clonal structure over narrow time frames via circulating tumor DNA in metastatic breast cancer. Molecular characterization of circulating tumour cells identifies predictive markers for outcome in primary, triple-negative breast cancer patients. Molecular evaluation of PIK3CA gene mutation in breast cancer: determination of frequency, distribution pattern and its association with clinicopathological findings in Indian patients.

Multiple signaling pathways involved in activation of matrix metalloproteinase-9 MMP-9 by heregulin-beta1 in human breast cancer cells. Multiplex detection of DNA mutations by the fluorescence fingerprint spectrum technique.

Mutation of the PTCH1 gene predicts recurrence of breast cancer. Mutation profiling identifies numerous rare drug targets and distinct mutation patterns in different clinical subtypes of breast cancers. Mutational characterization of individual breast tumors: TP53 and PI3K pathway genes are frequently and distinctively mutated in different subtypes.

N-substituted hydroxynaphthalene imino-oxindole derivatives as new class of PI3-kinase inhibitor and breast cancer drug: Molecular validation and structure-activity relationship studies. N-WASP activation by a beta1-integrin-dependent mechanism supports PI3K-independent chemotaxis stimulated by urokinase-type plasminogen activator. Neoadjuvant chemotherapy in breast cancer: a dose-dense schedule in real life and putative role of PIK3CA mutations.

Noninvasive detection of activating estrogen receptor 1 ESR1 mutations in estrogen receptor-positive metastatic breast cancer. Novel functional PI 3-kinase antagonists inhibit cell growth and tumorigenicity in human cancer cell lines. Novel mutations in neuroendocrine carcinoma of the breast: possible therapeutic targets. Oleate activates phosphatidylinositol 3-kinase and promotes proliferation and reduces apoptosis of MDA-MB breast cancer cells, whereas palmitate has opposite effects.

OPENchip: an on-chip in situ molecular profiling platform for gene expression analysis and oncogenic mutation detection in single circulating tumour cells. Osteopontin: it's role in regulation of cell motility and nuclear factor kappa B-mediated urokinase type plasminogen activator expression.

Particular molecular and ultrastructural aspects in invasive mammary carcinoma. Pathway-driven discovery of rare mutational impact on cancer. Pharmacological explorations of eco-friendly amide substituted Z -? Phospatidylinositol 3-kinase expression in human breast cancer. Phosphatidyl-inositolkinase alpha catalytic subunit mutation and response to neoadjuvant endocrine therapy for estrogen receptor positive breast cancer. Phosphatidylinositol 3-kinase and NF-kappaB regulate motility of invasive MDA-MB human breast cancer cells by the secretion of urokinase-type plasminogen activator.

Phosphatidylinositolkinase and AKT1 mutations occur early in breast carcinoma. Phosphatidylinositolkinase pathway mutations are common in breast columnar cell lesions. Phosphoinositide 3-kinase assay in breast cancer cell extracts. Phosphoinositide 3-kinase inhibition in the treatment of hormone receptor-positive breast cancer. Phosphoinositide 3-kinase inhibitors in advanced breast cancer: A systematic review and meta-analysis. Phosphoinositide 3-kinase mutations in breast cancer: a 'good' activating mutation?

Phosphoinositide 3-kinase signaling is critical for ErbB3-driven breast cancer cell motility and metastasis. Phosphoinositide 3-kinase signalling in breast cancer: how big a role might it play? Phosphoinositide 3-kinase targeting by the beta galactoside binding protein cytokine negates akt gene expression and leads aggressive breast cancer cells to apoptotic death.

Physiological levels of Pik3ca HR mutation in the mouse mammary gland results in ductal hyperplasia and formation of ER? PI3K inhibition enhances the anti-tumor effect of eribulin in triple negative breast cancer.

PI3K inhibition in breast cancer: Identifying and overcoming different flavors of resistance. PI3K inhibition results in enhanced estrogen receptor function and dependence in hormone receptor-positive breast cancer. PI3K inhibition to overcome endocrine resistance in breast cancer. PI3K mutations in breast cancer: prognostic and therapeutic implications. PIK3CA cancer mutations display gender and tissue specificity patterns.

PIK3CA exon 20 mutation is independently associated with a poor prognosis in breast cancer patients. PIK3CA exon 20 mutations are associated with poor prognosis in breast cancer patients. PIK3CA expression in invasive breast cancer: a biomarker of poor prognosis. PIK3CA gene is frequently mutated in breast carcinomas and hepatocellular carcinomas. PIK3CA gene mutations in the helical domain correlate with high tumor mutation burden and poor prognosis in metastatic breast carcinomas with late-line therapies.

PIK3CA mutation and histological type in breast carcinoma: high frequency of mutations in lobular carcinoma. PIK3CA mutation associates with improved outcome in breast cancer. PIK3CA mutation detected by liquid biopsy in patients with metastatic breast cancer.

PIK3CA mutation enrichment and quantitation from blood and tissue. PIK3CA mutation inhibition in hormone receptor-positive breast cancer: time has come. PIK3CA mutation is associated with a favorable prognosis among patients with curatively resected esophageal squamous cell carcinoma.

PIK3CA mutation predicts resistance to breast cancer therapy. PIK3CA mutation profiling in patients with breast cancer, using a highly sensitive detection system. PIK3CA mutational status in circulating tumor cells can change during disease recurrence or progression in patients with breast cancer. PIK3CA mutations and downstream effector p-mTOR expression: implication for prognostic factors and therapeutic targets in triple negative breast cancer.

PIK3CA mutations and their response to neoadjuvant treatment in early breast cancer: A systematic review and meta-analysis. PIK3CA mutations are a predictor of docetaxel plus epirubicin neoadjuvant chemotherapy clinical efficacy in breast cancer. PIK3CA mutations are associated with lower rates of pathologic complete response to anti-human epidermal growth factor receptor 2 her2 therapy in primary HER2-overexpressing breast cancer.

PIK3CA mutations are associated with reduced pathological complete response rates in primary HER2-positive breast cancer: pooled analysis of patients from five prospective trials investigating lapatinib and trastuzumab. PIK3CA mutations define favorable prognostic biomarkers in operable breast cancer: a systematic review and meta-analysis.

PIK3CA mutations early persistence in cell-free tumor DNA as a negative prognostic factor in metastatic breast cancer patients treated with hormonal therapy. PIK3CA mutations in androgen receptor-positive triple negative breast cancer confer sensitivity to the combination of PI3K and androgen receptor inhibitors.

PIK3CA mutations in breast cancer are associated with poor outcome. PIK3CA mutations in breast cancer: reconciling findings from preclinical and clinical data. PIK3CA mutations in ductal carcinoma in situ and adjacent invasive breast cancer. PIK3CA mutations in in situ and invasive breast carcinomas. PIK3CA mutations rarely demonstrate genotypic intratumoral heterogeneity and are selected for in breast cancer progression. PIK3CA mutations, phosphatase and tensin homolog, human epidermal growth factor receptor 2 and insulin-like growth factor 1 receptor and adjuvant tamoxifen resistance in postmenopausal breast cancer patients.

PIK3CA oncogenic mutations in neoadjuvant treatments for breast cancer. PIK3CA somatic alterations in invasive breast cancers: different spectrum from Caucasians to Chinese detected by next generation sequencing. PIK3R1 underexpression is an independent prognostic marker in breast cancer. PIKing the right isoform: the emergent role of the pbeta subunit in breast cancer. Precision cell-free DNA extraction for liquid biopsy by integrated microfluidics.

Precision medicine for metastatic breast cancer--limitations and solutions. Precision medicine for metastatic breast cancer. Preclinical modeling of combined phosphatidylinositolkinase inhibition with endocrine therapy for estrogen receptor-positive breast cancer. Predictive biomarkers of sensitivity to the phosphatidylinositol 3' kinase inhibitor GDC in breast cancer preclinical models. Prognostic factors in the myoepithelial-like spindle cell type of metaplastic breast cancer.

Prognostic role of PIK3CA mutations and their association with hormone receptor expression in breast cancer: a meta-analysis. Proximity ligation assay to detect and localize the interactions of ER? Radium dichloride bone-targeted alpha particle therapy for hormone-refractory breast cancer metastatic to bone. Ramentaceone, a Naphthoquinone Derived from Drosera sp.

Reconstructing kinase network topologies from phosphoproteomics data reveals cancer-associated rewiring. Regulation of breast cancer cell chemotaxis by the phosphoinositide 3-kinase pdelta. Regulation of fatty acid synthase expression in breast cancer by sterol regulatory element binding protein-1c. Regulation of MDR1 expression and drug resistance by a positive feedback loop involving hyaluronan, phosphoinositide 3-kinase, and ErbB2.

Regulation of miRNAc and its downstream pathways in preneoplastic progression of triple-negative breast cancer. Relationship of PIK3CA mutation and pathway activity with anti-proliferative response to aromatase inhibition. Resveratrol-induced apoptosis in MCF-7 human breast cancer cells involves a caspase-independent mechanism with downregulation of Bcl-2 and NF-kappaB. RNA interference-mediated depletion of phosphoinositide 3-kinase activates forkhead box class O transcription factors and induces cell cycle arrest and apoptosis in breast carcinoma cells.

Roles of BIM induction and survivin downregulation in lapatinib-induced apoptosis in breast cancer cells with HER2 amplification. Roles of human epidermal growth factor receptor 2, c-jun NH2-terminal kinase, phosphoinositide 3-kinase, and p70 S6 kinase pathways in regulation of cyclin G2 expression in human breast cancer cells. Science Signaling Podcast for 21 February Pentraxin-3 in basal-like breast cancer. Sensitive cytometry based system for enumeration, capture and analysis of gene mutations of circulating tumor cells.

Sensitization of breast cancer cells to radiation by trastuzumab. Serine phosphorylation of paxillin by heregulin-beta1: role of p38 mitogen activated protein kinase. Signaling-dependent nuclear export of estradiol receptor controls cell cycle progression in breast cancer cells.

Significant prognostic values of differentially expressed-aberrantly methylated hub genes in breast cancer. Single cell mutational analysis of PIK3CA in circulating tumor cells and metastases in breast cancer reveals heterogeneity, discordance, and mutation persistence in cultured disseminated tumor cells from bone marrow.

Single-cell resolved imaging reveals intra-tumor heterogeneity in glycolysis, transitions between metabolic states, and their regulatory mechanisms. Somatic mutation profiling in BRCA-negative breast and ovarian cancer patients by multigene panel sequencing.

Specific requirement for the ppalpha phosphatidylinositol 3-kinase during epidermal growth factor-stimulated actin nucleation in breast cancer cells. Structural prediction, whole exome sequencing and molecular dynamics simulation confirms p. Subclonal diversification of primary breast cancer revealed by multiregion sequencing.

Suppression of epidermal growth factor receptor-mediated? Surgical resection of breast cancers: Molecular analysis of cancer stem cells in residual disease. Survivin-targeting miRp overcomes HER3 signaling-induced chemoresistance and enhances the antitumor activity of paclitaxel against HER2-overexpressing breast cancer.

Syk, a protein-tyrosine kinase, suppresses the cell motility and nuclear factor kappa B-mediated secretion of urokinase type plasminogen activator by inhibiting the phosphatidylinositol 3'-kinase activity in breast cancer cells. Synergistic interactions with PI3K inhibition that induce apoptosis.

Systematic functional characterization of resistance to PI3K inhibition in breast cancer. Systematic identification of genes with a cancer-testis expression pattern in 19 cancer types. Targeted deep sequencing revealed variants in cell-free DNA of hormone receptor-positive metastatic breast cancer patients. Targeted RNAseq assay incorporating unique molecular identifiers for improved quantification of gene expression signatures and transcribed mutation fraction in fixed tumor samples.

Targeting 3-phosphoinoside-dependent kinase-1 to inhibit insulin-like growth factor-I induced AKT and p70 S6 kinase activation in breast cancer cells. Targeting HER2 heterogeneity in early-stage breast cancer. Targeting mTOR pathway inhibits tumor growth in different molecular subtypes of triple-negative breast cancers.

Testing considerations for phosphatidylinositolkinase catalytic subunit alpha as an emerging biomarker in advanced breast cancer. The anticancer activities of Vernonia amygdalina Delile. Leaves on 4T1 breast cancer cells through phosphoinositide 3-kinase PI3K pathway. The cancer genetics and pathology of male breast cancer.

The favorable impact of PIK3CA mutations on survival: an analysis of patients with breast cancer. The impact of heterogeneity in Phosphoinositide 3-Kinase pathway in human cancer and possible therapeutic treatments. The impact of PI3K inhibitors on breast cancer cell and its tumor microenvironment. The interaction of pCas with PKN3 promotes malignant growth. The molecular contribution of TNF-? The paradox of cancer genes in non-malignant conditions: implications for precision medicine.

The role of adapter protein Shc in estrogen non-genomic action. The role of insulin receptor substrate-1 in transformation by v-src. The role of phosphoinositide 3-kinase in breast cancer: an overview. Treating breast cancer through novel inhibitors of the phosphatidylinositol 3'-kinase pathway. Triplet therapy with palbociclib, taselisib and fulvestrant in PIK3CA mutant breast cancer and doublet palbociclib and taselisib in pathway mutant solid cancers.

Understanding the histogenesis of a HRAS-PIK3R1 co-driven metastatic metaplastic breast carcinoma associated with squamous metaplasia of lactiferous ducts. USP35, regulated by estrogen and AKT, promotes breast tumorigenesis by stabilizing and enhancing transcriptional activity of estrogen receptor?. XIAP over-expression is an independent poor prognostic marker in Middle Eastern breast cancer and can be targeted to induce efficient apoptosis.

Breast Neoplasms, Male. Activated phosphoinositide 3-kinase? RNAi screen of endoplasmic reticulum-associated host factors reveals a role for IRE1alpha in supporting Brucella replication. Burkitt Lymphoma.

Cross-linking of surface IgM in the Burkitt's lymphoma cell line ST provides protection against arsenite- and stress-induced apoptosis that is mediated by ERK and phosphoinositide 3-kinase signaling pathways. Hydrogen peroxide in the Burkitt's lymphoma cell line Raji provides protection against arsenic trioxide-induced apoptosis via the phosphoinositide-3 kinase signalling pathway. Immunocytochemical detection of phosphatidylinositol 3 kinase in Burkitt lymphoma cells.

A constitutively activated form of the pbeta isoform of PI3-kinase induces prostatic intraepithelial neoplasia in mice. A PIK3CA transgenic mouse model with chemical carcinogen exposure mimics human oral tongue tumorigenesis. Activated Ki-Ras suppresses O-tetradecanoylphorbolacetate-induced activation of the c-Jun NH2-terminal kinase pathway in human colon cancer cells.

Activation of PI3K-Akt signaling pathway promotes prostate cancer cell invasion. Akt isoform-specific signaling in breast cancer: uncovering an anti-migratory role for palladin. An activating Pik3ca mutation coupled with Pten loss is sufficient to initiate ovarian tumorigenesis in mice.

An insulin-like growth factor-mediated, phosphatidylinositol 3' kinase-independent survival signaling pathway in beta tumor cells. An integrated strategy for revealing the pharmacological changes based on metabolites profiling and network pharmacology: Arctiin as an example. B, a novel specific pan-PI3K inhibitor, preferentially targets cancer stem cells. Binding of ras to phosphoinositide 3-kinase palpha is required for ras-driven tumorigenesis in mice.

Biological functions of cytokeratin 18 in cancer. Characterization of chromosome 3q and 12q amplicons in nasopharyngeal carcinoma cell lines. Chemopreventive Effects of Licorice and Its Components. Class IA phosphoinositide 3-kinase isoforms and human tumorigenesis: implications for cancer drug discovery and development.

Co-activation of PIK3CA and Yap promotes development of hepatocellular and cholangiocellular tumors in mouse and human liver. Cribriform-morular variant of papillary thyroid carcinoma: a study of 3 cases featuring the PIK3CA mutation. Differential induction of apoptosis and inhibition of the PI3-kinase pathway by saturated, monounsaturated and polyunsaturated fatty acids in a colon cancer cell model. Endometrial cancer cells exhibit high expression of p?

Essential roles of PI 3 K-pbeta in cell growth, metabolism and tumorigenesis. Functional genomics reveals that tumors with activating phosphoinositide 3-kinase mutations are dependent on accelerated protein turnover. Gene expression profiling analysis contributes to understanding the association between non-syndromic cleft lip and palate, and cancer.

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Deep ocean coral reef adventure 720p official hd documentary torrent Precision cell-free DNA extraction dmba-155 torrent liquid biopsy by integrated microfluidics. Survival activity of troglitazone in rat motoneurones. The phosphoinositide 3-kinase pathway and cancer. Lung adenocarcinoma from East Asian never-smokers is a disease largely defined by targetable oncogenic mutant kinases. Organism related Information. Lack of phosphoinositide 3-kinase-gamma attenuates ventilator-induced lung injury. Lunasin, a novel seed peptide, sensitizes human breast cancer MDA-MB cells to aspirin-arrested cell cycle and induced apoptosis.


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